Primary subcutaneous nocardial infection in a SLE patient
Identifieur interne : 002646 ( Main/Exploration ); précédent : 002645; suivant : 002647Primary subcutaneous nocardial infection in a SLE patient
Auteurs : A. Balbir-Gurman ; D. Schapira ; A M Nahir [Israël]Source :
- Lupus [ 0961-2033 ] ; 1999-02.
English descriptors
- Teeft :
- Asteroides, Asteroides infection, Asteroides species, Blood cells, Brasiliensis, Case report, Childhood dermatomyositis, Cyclophosphamide, Cytotoxic, Diffuse proliferative glomerulonephritis, Erythematosus, Gadl, Immunosuppressive, Immunosuppressive therapy, Infection, Local trauma, Lupus, Lupus activity, Methotrexate, Mgad, Nocardia, Nocardia asteroides, Nocardia asteroides infection, Nocardia brasiliensis infection, Nocardial, Nocardiosis, Opportunistic, Opportunistic infections, Opportunistic organisms, Prednisolone, Primary nocardia skin infection, Primary subcutaneous nocardial infecction, Primary subcutaneous nocardiosis, Pulmonary nocardiosis, Rheumatoid, Rheumatoid arthritis, Rheumatol, Steroid, Subcutaneous, Systemic, Systemic lupus erythematosus, Typical gram.
Abstract
A patient with systemic lupus erythematosus (SLE) developed primary subcutaneous nocardiosis during steroid and cyclophosphamide therapy for diffuse proliferative glomerulonephritis. In spite of local process the patient manifested signs of general deterioration mimicking SLE exacerbation. The diagnosis was made by bacteriologic examination of the material obtained by CT guided aspiration. Surgical drainage and systemic treatment with trimetoprim/sulphamethoxazole (TMT/SMZ) 960 mg twice/d led to a clinical recovery and enabled the continuation of the steroid and cytotoxic regimen.
Url:
DOI: 10.1191/096120399678847542
Affiliations:
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Le document en format XML
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<term>Blood cells</term>
<term>Brasiliensis</term>
<term>Case report</term>
<term>Childhood dermatomyositis</term>
<term>Cyclophosphamide</term>
<term>Cytotoxic</term>
<term>Diffuse proliferative glomerulonephritis</term>
<term>Erythematosus</term>
<term>Gadl</term>
<term>Immunosuppressive</term>
<term>Immunosuppressive therapy</term>
<term>Infection</term>
<term>Local trauma</term>
<term>Lupus</term>
<term>Lupus activity</term>
<term>Methotrexate</term>
<term>Mgad</term>
<term>Nocardia</term>
<term>Nocardia asteroides</term>
<term>Nocardia asteroides infection</term>
<term>Nocardia brasiliensis infection</term>
<term>Nocardial</term>
<term>Nocardiosis</term>
<term>Opportunistic</term>
<term>Opportunistic infections</term>
<term>Opportunistic organisms</term>
<term>Prednisolone</term>
<term>Primary nocardia skin infection</term>
<term>Primary subcutaneous nocardial infecction</term>
<term>Primary subcutaneous nocardiosis</term>
<term>Pulmonary nocardiosis</term>
<term>Rheumatoid</term>
<term>Rheumatoid arthritis</term>
<term>Rheumatol</term>
<term>Steroid</term>
<term>Subcutaneous</term>
<term>Systemic</term>
<term>Systemic lupus erythematosus</term>
<term>Typical gram</term>
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<front><div type="abstract" xml:lang="en">A patient with systemic lupus erythematosus (SLE) developed primary subcutaneous nocardiosis during steroid and cyclophosphamide therapy for diffuse proliferative glomerulonephritis. In spite of local process the patient manifested signs of general deterioration mimicking SLE exacerbation. The diagnosis was made by bacteriologic examination of the material obtained by CT guided aspiration. Surgical drainage and systemic treatment with trimetoprim/sulphamethoxazole (TMT/SMZ) 960 mg twice/d led to a clinical recovery and enabled the continuation of the steroid and cytotoxic regimen.</div>
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<name sortKey="Schapira, D" sort="Schapira, D" uniqKey="Schapira D" first="D" last="Schapira">D. Schapira</name>
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<country name="Israël"><noRegion><name sortKey="Nahir, A M" sort="Nahir, A M" uniqKey="Nahir A" first="A M" last="Nahir">A M Nahir</name>
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